Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. On the other hand, our capability to rescue SERT function in KI midbrain synaptoneurosomes from the inhibition of FAK signifies elevated FAK signaling downstream of your Pro32Pro33 mutant, as confirmed by amplified pFAK localization in https://robertz087epa8.glifeblog.com/profile